INFECTOLOGÍA
Dr.. Christ Dr Christiam iam Oc Ochoa hoa
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COCOS GRAM POSITIVOS AEROBIOS CAT CATALASA ALASA PO POSIT SITIV IVOS OS
CAT CATALAS ALASA A NEGA NEGATIVOS TIVOS
estafilococus
Enterococus, streptococus
BACILOS GRAM POSITIVOS AEROBIOS Actinomicetos Actinomicetos con acidos micolicos: corynebacterium, corynebacterium, nocardia, mycobacterium.
Actinomicetos sin acidos micolicos: actinomicetos, actinomicetos, streptomyces.
OTROS: bacillus, erysipelotrix, listeria, gardnerella.
COCOS, COCOBACILOS Y BACILOS BA CILOS GRAM NEGA NEGATIVOS TIVOS AEROBIOS COCOS Y COCOBACILOS: moraxella, neisseria. BACILOS: enterobacteriaceae: citrobacter, enterobacter, escherichia, klebsiella, proteus, salmonella, serratia, shigella,
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ESTAFILOCOCOS ASPECTOS GENERALES GENERA LES • • • • • •
Todos son catalasa catalas a positivos . Coagulasa +: aureus aureus (manitol (manitol +) +) Coagulasa - : epidermidis epidermidis (manitol (manitol -) y saprophyticus saprophyticus (manitol +) Saprofitos y patógenos patógenos de la piel. Epidermidis el mas fc de prótesis y hospotalarias. Saprofiticus: Itu jóvenes.
PATOGENICIDAD: Pared – enximas – exotoxinas. INFECCIONES SISTEMICAS : Foli Folicu culi liti tis. s.
INFECCIONES SUPERFICIALES :
Impe Impeti tigo go ampo ampoll llos oso. o. Forunculosis (ántrax). Hidrosadenitis. Hidrosadenitis. Neumonia – osteomielitis – artritis endocarditis
EST ESTAFILO AFILOCOC COCOS OS
Mucocu Mucocutan tanea eas, s, sisté sistémic micas, as, toxin toxinas. as.
ENOCARDITIS INFECCIOSA COCO GRAM + AEROBIO
BACILOS GRAM + AEROBIO
ESTREPTOCOCOS PYOGENES
Faringoamigdalitis Faringoamigdalitis y cutáneas.
ST. ST. AGAL AGALAC ACTI TIAE AE
Gine Gineco colo logi gica cass y neon neonat atal ales es
NEUM NEUMOC OCOC OCOS OS
Neum Neumon onia ia,, meni mening ngit itis is,, otit otitis is,, sinu sinusi siti tis. s.
OTROS OTROS ESTREPT ESTREPTOCO OCOCOS COS
Faecalis Faecalis,, bovis, viridans viridans
BACI BACILL LLUS US ANTH ANTHRA RACI CISS
Carb Carbun unco co
CORYNEBACTERIUM DIFTERIAE
Difteria
LYSTERIA
Listeriosis
NOCARDIA
Nocardiosis
ERYSIPELOTRIX
erisipeloide
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ESTREPTOCOCOS
•
ASPECTOS GENERALES GENERA LES • •
•
•
DIFTERIA
Coco+, Coco+, anaerobi anaerobio o facultat facultativo, ivo, catalasa catalasa -. DIPLOCOCO. Beta Beta hemo hemolí líti tico cos s (tot (total al), ), alfa alfa (par (parci cial al)) gamma gamma (no hemol hemolít ítico icos). s). Clasif Clasifica icacio cion n Lancefiel Lancefield: d: A,B…O Proteina M: antifagocitico, antifagocitico, especifica especifica para cepa cepas s de grup grupo o A, el más más viru virule lent nto. o. Capsula. Enzimas. Toxinas: estreptolisinas (hemolisina O, antigénica y hemolisina S), eritrogenica (escarlatina).
•
•
•
GRUPO A (S. PYOGENES): PYOGENES): faringoamigadal faringoamigadalitis itis e infecciones cutá cutáne neas as esca escarl rlat atin ina, a, piod pioder erma ma (eri (erisi sipe pela la,, impé impéti tigo go estreptocócico, celulitis). GRUPO B (S. AGALACTIAE): sepsis puerperal, corioamnionitis, corioamnionitis, aborto septico. NEUM NEUMOC OCOC OCO O (alf (alfa a hemo hemolí líti tico co sens sensib ible le a opto optoqu quin ina) a).. Serotipos según polisacárido capstular. GRUPO D (gammahemoliticos): (gammahemoliti cos): Enteroco Enterococo co (faecali (faecalis) s) crece en NaCl: NaCl: ITUs por MAF, • intrumentacion, intrumentacion, edad media. AMPI+GENT AMPI+GENTA. A. No enterococo (bovis) no crece: Endocarditi Endocarditis s + ca colon • (>80% casos). Penicilina. ST. ST. VIRIDAN VIRIDANS: S: alfahemo alfahemoliti litico. co. Salivari Salivarius, us, mutans. mutans. Caries. Caries. ENDOCARDITIS SUBAGUDA NATIVA NATIVA (+fc).
ACTINOMICET A CTINOMICETOS OS
ERYSIPELOTHRIX
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• • •
LISTERIA •
Flora digestiva normal. Neonatos o ancianos. Transmi Transmite te por alimento alimentos s en brotes brotes (vegetale (vegetales, s, lácteos lácteos y carnes). Perinatal: meningitis post postpa part rto o 1 a 2 sema semana nas. s. Congenita: abscesos diseminados (granulomatosis (granulomatosis infantisepticum). infantisepticum). Ancianos o inmunosupromidos: bacteremia sin foco o meningitis. Dx cultivo. • Tto: penicilina O • AMPICILINA. AMPICILINA.
ANA A NAEROBIOS EROBIOS - CLOSTRIDIUM CL OSTRIDIUM COCOS GRAM +
Estreptococos: peptostreptococ os, peptococcus
BACILOS ESPORULADOS: ESPORULADOS: Clostridium Cl ostridium (palillo tambor)
NO ESPORULADOS: Actynomices, propionilbacterium, bifidobacterium. GRAM -
Veilonella, Megaspora
BACTEROIDES: BACTEROIDES: fragilis. FUSOBACTERIUM.
INVASIVOS: Perfringes y septicum. •
Gangrena gaseosa. Toxina Toxina alfa y M toxina (gas). TTO: Qx, penicilina, clindamicina, O2, antitoxina.
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ACTINOMYCES A CTINOMYCES ISRAELLI Hifas gram + anerobio. A. israelli. Absceso cervicofacial. Uso DIU. Granos de azufre.
OTROS OTRO S ANAEROBIOS ANA EROBIOS Cientos no esporulados. Flora comensal piel, colon, colon, boca, tracto tracto femenino femenino.. Estreptoc Estreptococos ocos anaerobios y B. melaninogenicus supradiafragmatico. B.fragilis y Fusobacterium
COCOS GRAMNEGATIVOS AEROBIOS 1. NEISSERIA NEISSERIA GONORRH GONORRHOEAE OEAE Y MENINGITIDI MENINGITIDIS. S. 2. MORAXELLA LACUNAT LACUNATA (conjuntivitis angular de Axenfeld) Y CAT CATARRHALIS (tercera de OMA despues de Neumococo y HIB) N. gonorrhoeae: Coco gonorrhoeae: Coco gram – aer aerobio obio,, inmó inmóvi vil, l, oxida xidasa sa positivo y maltosa negativo. 1. DIPL DIPLOC OCOC OCO O. Tipi Tipifi fica cado do por proteína I, se fijan por pili y proteína OPA. HUMANO UNICO
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B A CILOS GRAM NEGATIVOS NEGATIVOS ASPECTOS GENERALES GENERA LES Aerobios o anaerobios facultativos. Fermentativos. Fermentativos. Oxidasa
ENTEROBACTERIAS : 1. COMENS COMENSALE ALESS: E.coli, Klebsiella, Proteus,
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ETIOLOGIA DEFINICIÓN Es un tipo de Fiebre entérica
• •
Salmonella es un gram negativo, móviles y no esporuladas. La salmonella tiene antígenos O: somáticos, H: flagelares y Vi: de virulencia
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HAEMOPHILUS H.influenzae, ducreyi, aegypticus, parainfluenza.
LEGIONELLA Bacilo Bacilo Gram Gram negati negativo vo aerobio, móviles. Intracelulares al infectar.
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BARTONELLA ASPECTOS GENERALES GENERA LES
B. Henseale B. Quintana B. Bacilliformis
LEPTOSPIRA
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ETIOLOGIA Y CICLO BIOLOGICO EPIDEMIOLOGIA •
•
•
End Endem emic ico o en Mexi Mexico co,, America Central y Sur Mas Mas rie riesgo en zonas rurales Trasmisión: Trasmisión: contacto contacto con las las hece eces del vect vector or triatomo, tbn por: congénita, transfusiones, transplantes o accidente de laboratorio
T. BruceiT afric (enfer Sueño) T. Cruzi T ameri (enfer Chagas)
Tiene 3 estadios biológicos: • Tripomastigote: Tripomastigote: Movil, 20um. Es la forma infectante. infectante. Esta Esta en las heces del vector y en sangre del humano. • Amastigote: Amastigo te: Celula Celula redonda, 2-4um. Esta en el intracelular. • Epimastigote: Mas largo largo que el tripomastigote. Esta en el intestino intestino del vector. vector.
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ENFERMEDAD AGUDA • • • •
•
•
Periodo de de incubación: incubación: Aprox Aprox 1 semana Puede durar durar de 2ss 2ss a meses Puedes ser ser asintomático asintomático o sintomático sintomático Sintomas: Sintomas: signos signos del sitio de inoculación inoculación (Romaña o Chagoma), fiebre, adenopatías, edemas. Raro formas formas graves: graves: Miocarditi Miocarditiss aguda o MEC (lo peor) Se encuentra encuentra al al parasito parasito en sangre.
DIAGNOSTICO FASE AGUDA Identificar el • tripomastigo tripomastigote te por Gota Gota Grue Gruesa sa con con Giemsa FASE CRONICA • ELISA
FASE CRONICA INDETERMINADA •
Fase asintomática asintomáti ca parasitos
TRATAMIENTO
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T. gondii: huésped gondii: huésped definitivo gato. Ooquiste en heces – aves o mamíferos. Esporozoito se divide intracelularmente – taquizoitos taquizoitos (clínica (clínica y congenita) congenita).. Bradizoit Bradizoitos os (cronific (cronificaa – VIH reactiva/granulomas).
TOXOPLASMOSIS
•
Ingesta de carne mal cocida con quistes o por ooquistes del gato. Clinica:
CONGENIT CONGENITA: A: asintomá asintomática, tica, aborto, ictericia, ictericia, corioreti corioretinitis nitis (mas fc), Rm , hepatoesp hepatoespleno. leno. TETRADA SABIN: corioretinitis, hidrocefalia, calcificaciones intracraneales, convulsiones. • AGUDA AGUDA INMUNOCO INMUNOCOMPET MPETENTE ENTES: S: asintomáti asintomática, ca, linfadeno linfadenopatia patiass (linfaden (linfadenitis itis supurada supurada de Piringer-Kuchinka), Piringer-Kuchinka), retinocoroiditis leve. INMUNODEPRIMIDOS INMUNODEPRIMIDOS CELULAR: sepsis, meningoencefalitis meningoencefalitis (VIH). •
Dx:
1. 2. 3. Tto:
Ig-G 4x o IgM IgM o PCR PCR en agudos. agudos. IgG por por IFI, IFI, ELISA ELISA o dye-test dye-test (Sabin-Feldman) (Sabin-Feldman) en crónicos. crónicos. IgA o IgM IgM ELISA ELISA en en congén congénitas. itas. PIRIMETAMINA + SULFADIACINA (clinda, atovacuona, dapsona, CTX, claritromicina) + ACIDO
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NEMAT INTEST: Trichuris trichiura Tamaño: 4 Tamaño: 4 cm Color: rojizo •Color: rojizo Habitat: Colon, preferentemente en el ciego el ciego.. •Habitat: Colon, infectante: Huevo embrionados •Forma infectante: Huevo Transmision: via oral por fecalismo •Transmision: via •Porción cefálica filiforme y caudal gruesa. •
Tricocefalosis Tricocefalosis o tricuriasis Hacen patología patología por por lesión mecánica. inflamación local, edema y hemorragia. • La La gravedad de la patología es proporcional al # de parásitos. Clinica: depende del grado de infestacion: •
Infestaciones:
Leve: asintomaticos Leve: asintomaticos •Moderada: dolor Moderada: dolor tipo cólico y diarrea ocasional Severa: por parasitismo intenso y es •Severa: por grav grave, e, + fr en niño niñoss desn desnut utrid ridos os.. Diarrea disenterica colico •
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NEMAT INTEST: Enterobius Vermicularis Tamaño: Macho: Tamaño: Macho: 4mm Hembra: 10 – 12mm Color: blanco
• •
Oxiuriosis o enterobiosis El hombre es el único que sufre esta
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NEMAT INTEST: Ascaris lumbricoide Tamaño: Muy grande de 20 – 40cm Color: Blanco o rosado Hábitat: Intestino Delgado(Yeyuno) sin adherirse
Ascaridiasis Primera Infección por helmintos + fr en el mundo Clínica Respiratorios:
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NEMAT INTEST: Strongyloides stercoralis • •
Tamaño: 2mm Hábitat: + fr en Intestino Delgado
Estrongiloidiasis
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NEMAT INTEST: Strongyloides stercoralis Estrongiloidiasis
Tienes cuadros clínicos como:
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NEMAT INTEST: Ancyostoma duodenale y Necator americanus • • •
Tamaño: 10 mm Color: Blanquecinos o herrumbrosos Habitat: INTESTINO DELGADO
Uncinariasis Es la segunda
infeccion
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VIRUS VIRU S EBSTEIN-BARR EBSTEIN-BA RR
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CMV
El CMV CMV se adqu adquie iere re a través de la sangre, el tejido y la mayoría de secreciones corp orporal oralees. El CMV CMV provoc provocaa una infecc infección ión prod produc uctiv tivaa de célu célula lass epiteliales y de otro tipo El CMV establece latencia en los linfocitos T, macróf macrófago agoss y otras otras célula células. s. La inmuni inmunida dad d mediada por células es necesaria para la ación, ación, a la
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ETIOLOGIA Y VECTOR DEFINICIÓN
• •
Enfermedad Enfermedad viral aguda endemo-epidémica. endemo-epidémica. •
EPIDEMIOLOGIA • •
•
Es de notificación obligatoria La vigilancia vigilancia se hace con las definici definiciones ones de casos: CP sin signos de alarma, CP con signos de alar alarma ma,, CP de deng dengue ue gra grave, ve, CC por por labor laborato atorio rio,, CC por nexo nexo epide epidemio miológ lógico ico,, Caso descartado. FACTORES DE RIESGO
•
• • •
Virus del Dengue Familia Togaviridae Togaviridae Genero Genero Flavivirus. ARN de cadena cadena simple (monocatenario) El virus tiene 3 proteínas estructurales (E, (E, M, C), 1 ARN y proteínas no estructurales NS1, NS2A, NS2B, NS3, NS4A, NS4B, NS5. 4 serotipos: DEN-1,2,3,4. DEN-1,2,3,4. Estos serotipo serotipo son por por variación de la Proteina E4. El 2 y 3 mas asociado a formas graves. Hay cierta inmunidad cruzada. No hay hay reservorio animal, se cree que algunos monos. Trans ransmi misi sión ón:: Cicl Ciclo o natu natura rall se da entr entree el homb hombre re y el mosquito Aedes aegypti.
CLINICA
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DIAGNOSTICO • •
Sospecha clínica y epidemiologica Diagnostico especifico: 1. ELISA ELISA IgM, IgM, IgG (de elecció elección) n) 2. Detecc Deteccion ion del Ag NS1 3. Detecc Detección ión de ARN ARN viral viral por PCR
TRATAMIENTO
1. 2.
3.
Repos Reposo o e Hidr Hidrat atac ació ión n Sint Sintom omát átic ico. o. • Dar paracetamol NO aspirina. • No hay hay vacu vacuna na..
Caso de Dengue sin signos de Alarma (GRUPO A): Estos pacientes pueden ser manejados en el hogar, si toleran volúmenes adecuados de líquidos por la vía oral y han orinado por lo menos una vez cada 6 horas, no tienen signos de alarma y se descarta en
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ETIOLOGIA Y VECTOR DEFINICIÓN • • •
Enfermedad febril aguda aguda de corta duración duración Es una zoonosis
EPIDEMIOLOGIA
• • •
•
• • • •
Es una enfermedad de declaración declaración mundial mundial Tiene vacuna vacuna y esta en en el esquema Endémico, +fr varones varones y adultos agricultores agricultores 3 ciclos ecológicos, (identificados en Africa) Africa) • Ciclo selvático es selvático es por el mosquito Haemagogus en América Latina y en África por Aedes Aedes africanus. • Ciclo rural, rural, es por el mosquito Aedes simposi (en Africa) • Ciclo urbano, urbano, es el mismo en América y África por Aedes
Virus de la Fiebre Fiebre Amarilla Familia Togaviridae Togaviridae Genero Genero Flavivirus. ARN de cadena cadena simple (monocatenario) (monocatenario) Tiene 1 solo serotipo El vector es el Aedes Aedes agypti son mosquitos mosquitos diurnos que abundan abundan en zonas zonas humedad alrededor de aguas estancadas . Nicho ecológico ecológico en Perú: Perú: Rupa-Rupa Rupa-Rupa
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DIAGNOSTICO
TRATAMIENTO • •
No tiene manejo manejo especifico Mantenimiento
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VIH •
•
Gen env gp41, gp120 • envoltura Gen gag p18, p24, p15 •
VIH-1
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COMPROMISOS NEUROLOGICOS EN VIH
NEUROTOXOPLASMOSIS Sindrome de focalizacio neurologica o convulsiones. NEUROCRIPTOCOCOCIS Sindrome de Hipertension Endocraneana Endocraneana MEC TBC Sindrome Meningo-Encefalitico Meningo-Encefalitico DEMENCIA Leucoencefalopatia Leucoencefalopatia Multifocal Progresiva (LEMP)
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